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I can judge Iressa's effectiveness on my personal experience. I used the medication after having undergone my intravenous chemotherapy. I started taking Iressa in 2005. My last scan after the chemotherapy and Iressa treatment showed that the cancer stopped growing. I also noticed the improvement in my condition; I started feeling myself much better than before. Each scan thereafter showed slow but still noticeable improvement. I'm still taking Iressa and still showing signs of improvement. Wonderful medication for lung cancer.

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Conclusion EGFR gene copy number was a predictor of clinical benefit from gefitinib in ISEL.

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Surgery may be performed before or after other forms of treatment.

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It is not a chemotherapeutic in the traditional sense, since it does not cause the destruction of tumor cells.

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Chapter Six - The Ubiquitin Network in the Control of EGFR Endocytosis and Signaling.

Growth inhibition is expressed as the percentage of surviving cells in drug-treated versus PBS-treated control cells (which is considered as 100% viability).

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Note: In clinical trials, multiple peripheral infusions given through the same vein resulted in infusion-site reactions.

NSCLC patients with an activating EGFR mutation, initial treatment with an EGFR tyrosine kinase inhibitor (TKI) was superior to standard platinum-based chemotherapy.

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PD model between plasma drug concentration and the corresponding tumor PD response, in this case pEGFR ().

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Surgery is the treatment of choice for patients with stage II NSCLC.

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Immunohistochemical analysis of paraffin-embedded human breast carcinoma, control (left) or λ phosphatase-treated (right), using Phospho-PRAS40 (Thr246) (C77D7) Rabbit mAb.

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Although the current evidence is conflicting, FDG-PET scanning may improve results of early-stage lung cancer by identifying patients who have evidence of metastatic disease that is beyond the scope of surgical resection and that is not evident by standard preoperative staging procedures.

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